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Il Cancro nasce in
sintesi, secondo la
Medicina naturale perche' l'organismo del
canceroso e'
intossicato,
infiammato,
immunodepresso, con
flora batterica alterata,
pH digestivo non regolare (e quindi mancante di
minerali e
vitamine per malnutrizione), e parassitato da certi,
parassiti,
batteri e
funghi (candida) i quali producono
tossine ed ulteriori
infiammazioni, ma e' "gestito",
cioe' Causato dai
Conflitti Spirituali (consci ed inconsci)
e dall'intenso
stress.
Esso e' quindi una malattia
MULTIFATTORIALE.
Quindi il medico, il terapeuta od il soggetto stesso
DEVE operare seguendo ed invertendo, la stessa strada percorsa per
l'ammalamento.
Cioe' deve lavorare per
disintossicare il
malato + disinfiammare l'organismo ed i
tessuti interessati, ripristinare il
pH digestivo,
eliminare quei parassiti, batteri e funghi
che hanno proliferato in modo abnorme, per mancanza
di antagonisti e rinforzare
il sistema immunitario
SEMPRE
compromesso in TUTTI i malati, cancerosi
compresi ed eliminare il Conflitto spirituale e lo
stress esistenti.
>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>
Il
TERRENO ONCOLOGICO (ed
e' valutabile da parte di medici
appositamente
istruiti) esiste, ma gli informatori, come
Repubblica-Salute, ed altre riviste importanti a
livello internazionale... lo ignorano,
malgrado siano state
informate !
Ecco le prove che il
Terreno Oncologico esiste:
Vegf
senza controllo
Pare che
siano 2 le mutazioni che compromettono la corretta
inibizione dell'ormone. Così si sviluppa il tumore
più diffuso fra i bambini. Lo studio è pubblicato su
Nature Medicine
Ricercatori belgi e statunitensi hanno scoperto il
meccanismo alla base dell'emangioma, il tumore più
comune fra i bambini.
Questa neoplasia, che in Europa colpisce il 10 per
cento dei neonati, prolifera nei tessuti dei vasi
sanguigni e li danneggia irreversibilmente. Il
tumore si sviluppa velocemente fin nei primi giorni
di vita e, in molti casi, rallenta nei mesi
successivi fino a scomparire del tutto verso la fine
della pubertà. Sebbene si presenti in forma benigna,
la malattia può avere gravi conseguenze come
malformazioni e disfunzioni.
Lo studio - condotto da un gruppo di ricercatori di
Harvard, del
Children's Hospital di Boston e del
Christian de Duve Insitute di Lovanio (Belgio),
e pubblicato questa settimana su
Nature Medicine - ha identificato la causa della
proliferazione incontrollata delle cellule.
Nel tessuto sano, sulla superficie della cellula è
presente un recettore che ha il compito di inibire
l'azione dell'ormone Vegf (fattore di crescita
vascolare endoteliale), che altrimenti le darebbe
l’ordine di moltiplicarsi. L’analisi dei tessuti
prelevati da nove diversi pazienti ha permesso di
individuare due mutazioni genetiche che
compromettono il funzionamento del recettore,
lasciando libero l’ormone di dare avvio alla
proliferazione cellulare anomala. I ricercatori
hanno inoltre osservato che il tumore si origina
sempre all’interno del tessuto endoteliale, lo
strato che riveste la parete interna del vaso
sanguigno
MicroRna: verso una nuova terapia
Testati in modeli animali, MiR-15a e miR-16-1
sembrano in grado di bloccare lo sviluppo del
cancro. La ricerca italiana su Nature Medicine
Due piccoli frammenti di Rna sembrano in grado di
arrestare il progresso del tumore alla prostata. Si
chiamano miR-15a e miR-16-1 e sopprimerebbero
l’attività di diversi oncogeni.
La scoperta si deve a un gruppo di ricercatori
italiani dell’Istituto
superiore di sanità (Iss) e dell’Istituto
oncologico del Mediterraneo coordinati da
Ruggero De Maria, ed è stata pubblicata su
Nature Medicine.
Analizzando i tessuti tumorali di 40 pazienti con
cancro alla prostata, i ricercatori hanno notato che
una piccola porzione di DNA, normalmente localizzata
sul tredicesimo cromosoma, non era presente.
In questa regione si agganciano solitamente due
microRna (miR) non codificanti (che non portano cioè
alla formazione di alcuna proteina), miR-15a e
miR-16-1 per l’appunto, su cui si è concentrato lo
studio.
Gli esperimenti effettuati sui topi hanno rivelato
che la scomparsa dei due frammenti di Rna promuove
la trasformazione delle cellule prostatiche in
tumorali e accelera la loro proliferazione.
Introdurre nuovamente nelle cellule cancerose i
microRna, invece, blocca questa crescita e distrugge
le cellule cancerose stesse, facendo regredire la
malattia. MiR-15a e miR-16-1, secondo lo studio,
sarebbero legati all’attività di tre oncogeni, Bcl2,
Ccnd1 e Wnt3a, la cui espressione infatti aumenta
nei casi di tumore alla prostata.
Secondo i ricercatori, la scoperta potrebbe
agevolare lo sviluppo di nuove terapie e rivelarsi
utile nella diagnosi precoce: i medici potrebbero
valutare l’aggressività del cancro prostatico
proprio grazie alla presenza o assenza di miR-15a e
miR-16-1. (a.g.)
Tratto da: Galileonet.it
>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>
Dear Colleagues,
I read your fascinating article, published on the
"elite" peer-review Science, open to papers coming
ONLY from Univeristy centres....
However, I ask me what help will be it for
General Practitioners, who certainly know since two
decades... that there are many other gaseous
signaling molecules in biology and medicine, beside
"radical" NO (if active, NO must be RADICAL: Am I
right?).
In my opinion, the present Mean Age of Medicine will
continue as far as ELITE peer-reviews overlook such
as advinces in CLINICAL field, like the following,
really NOT RECENT !
WHAT ACCOUNT FOR THE REASON arterial compliance is
impaiered, is the INHERITED alteration of
endothelial function!
In addition, as s regards Hypertension
pho-physiology we must consider both Hypertension
Biophysical Semeiotic Constitution and hypertensive
Inherited Real Risk, YOU and Science Editors and
Reviers Unfortunately ignore !
See
www.semeioticabiofisica.it +
http://www.mednat.org/cancro/terreno_oncologico.htm
But not
www.nature.com, Thanks God.
for instance, See:
http://blogs.nature.com/reports/theniche/2008/04/stemcell_skin_creams_a_san_die.html#comments
http://www.nature.com/news/2008/080130/full/451511a.html?q=2#last-comment
http://www.washingtonpost.com/ac2/wp-dyn/comments/display?contentID=AR2007041101736&start=41
http://www.nature.com/news/2008/080822/full/news.2008.1060.html?q=2#last-comment
http://blogs.nature.com/reports/theniche/2008/06/pfizer_dips_a_toe_into_cell_th.html#comments
http://www.nature.com/news/2008/080604/full/news.2008.875.html?q=2#last-comment
http://blogs.nature.com/news/thegreatbeyond/2008/09/obama_speaks_on_science.html#comments
http://blogs.nature.com/news/thegreatbeyond/2007/09/californias_new_stem_cell_supr.html
Based on my 52-year-long
clinical, well-established experience I state that
the evaluation of endothelial function, in every
biological system, is realized nowadays also
clinically with a simple stethoscope, thanks to
Quantum Biophysical Semeiotics (1-10).
Unfortunately, almost
all authors as well as editors (especially those of
the famous Science!), ignore or overlook a reliable
clinical tool useful in bedside evaluating easily
and rapidly endothelial cell functions: Biophysical
Semeiotics
See
http://www.semeioticabiofisica.it/microangiologia.it.
In fact, among different techniques to evaluate the
endothelium functional capacity, that depend on the
amount of “radical” NO, as well as hydrogen sulfide
(H2S), physiologically generated by
cystathionine-lyase (CSE), and the vasodilatation
effect, nowadays there are biophysical-semeiotic
methods, applicable on very large scale in a few
minutes, even under stress tests. In fact, doctors
may nowadays bedside assess the synthesis intensity
of endothelial cell “radical” NO, as well as
hydrogen sulfide (H2S), physiologically generated by
cystathionine  -lyase (CSE), in order to recognize both
artery compliance alteration and ALL other disorders,
in simple way (1-5). The percentage of vasodilation
immediately after Valsalva’s Manoeuvre, confronting
it with the basal value, represents the endothelial
functional capacity, that is in relation to the
local parenchymal cell functions, according to my
Angiobiopathy theory, which complete that of
Tischendorf's Angiobiotopy (11).
Taking into account that shear stress is one of the
most important stimulants for the synthesis and
release of “radical” NO and H2S, the non invasive
technique most often used is the transient
flow-modulate “endothelium-dependent” post-ischemic
vasodilatation, performed on conductance arteries
such as the brachial, radial or femoral arteries.
Certainly shear-stress stimulates e-NO and H2S
synthesis in healthy individual, as allows me to
state Biophysical Semeiotics (1-5, 8-10). In
addition doctors gather a lot of other information,
e.g., after drugs administration (4-8). In fact,
such as vasodilatation is compared with the
vasodilatation produced by drugs that are NO and
H2S donors, such as nitroglycerine, called
“endothelium independent”.
The vasodilatation is quantified by measuring the
arterial diameter with high resolution
ultrasonography. Laser-Doppler techniques are now
starting to be used that also consider tissue
perfusion. One must admitt that such as methods
cannot be applied neither in apparently "healthy"
individual in order to perform primary prevention,
neither on very large scale even in diseased
subjects. All that accounts for the reason that
Microcirculation and Clinical Microangiology under
both physiological and pathological conditions, are
scarsely known by physicians all around the world,
also at universities, unfortunately.
Finally, thanks to
Quantum Biophysical Semeiotics, as well as the
unknown knowledge of non local realm in biological
systems beside the local realm, I recently
demonstrated (12-15), doctors are able in 1 second
to bedside exclude macro- and micro-vessels
alteration: in health, “intense” digital pressure,
applied upon an artery do not bring about
simultaneously aspecific gastric reflex, whereas
such as reflex appears in presence of whatever
vascular disorder Inherited Real Risk in individuals
involved by well-defined Biophysical-Semeiotic
Constitutions (12-14)
References:
1) Stagnaro-Neri
M., Stagnaro S. Introduzione alla Semeiotica
Biofisica. Il Terreno Oncologico. Travel Factory,
Roma, 2004.
http://www.travelfactory.it
2) Stagnaro S.,
Stagnaro-Neri M., Basi microcircolatorie della
semeiotica biofisica. Atti del XVII Cong. Naz. Soc.
Ital. Studio Microcircolazione, Firenze ott. 1995,
Biblioteca Scient. Scuola Sanità Militare, 2, 94.
3) Stagnaro-Neri M., Stagnaro S., Semeiotica
Biofisica: valutazione della compliance arteriosa e
delle resistenze arteriose periferiche. Atti del
XVII Cong. Naz. Soc. Ital. Studio Microcircolazione,
Firenze Ott. 1995, Biblioteca Scient. Scuola Sanità
Militare, 2, 93, 1995.
4) Stagnaro-Neri M., Stagnaro S., Semeiotica
Biofisica: valutazione clinica del picco precoce
della secrezione insulinica di base e dopo
stimolazione tiroidea, surrenalica, con glucagone
endogeno e dopo attivazione del sistema
renina-angiotesina circolante e tessutale – Acta Med.
Medit. 13, 99, 1997.
5) Stagnaro-Neri M., Stagnaro S., Semeiotica
Biofisica: la manovra di Ferrero-Marigo nella
diagnosi clinica della iperinsulinemia-insulino
resistenza. Acta Med. Medit. 13, 125, 1997.
6) Stagnaro-Neri M., Stagnaro S., Sindrome di Reaven,
classica e variante, in evoluzione diabetica. Il
ruolo della Carnitina nella prevenzione del diabete
mellito. Il Cuore. 6, 617 , 1993[Medline]
7) Stagnaro
Sergio Endothelial cell function can ameliorate
under safer drugs, such as Melatonin-Adenosine. BMC
Cardiovascular disorders, 10 October 2004.
http://www.biomedcentral.com/1471-2261/4/4/comments
8) Stagnaro S.,
Percussione Ascoltata degli Attacchi Ischemici
Transitori. Ruolo dei Potenziali Cerebrali Evocati.
Min. Med. 76, 1211, 1985 [Medline]
9) Stagnaro-Neri M., Stagnaro S., La sindrome
percusso-ascoltatoria da carenza di Carnitina. Clin.
Ter. 145, 135, 1994 [Medline]
10) Stagnaro-Neri M., Stagnaro S., Sindrome di
Reaven, classica e variante, in evoluzione
diabetica. Il ruolo della Carnitina nella
prevenzione del diabete mellito. Il Cuore. 6, 617 [Medline]
11)
Stagnaro Sergio. Role of Coronary Endoarterial
Blocking Devices in Myocardial Preconditioning - c007i.
Lecture, V Virtual International Congress of
Cardiology.
http://www.fac.org.ar/qcvc/llave/c007i/stagnaros.php
2007
12) Stagnaro Sergio e Paolo Manzelli.
Semeiotica Biofisica: Realtà non-locale in Biologia.
Dicembre 2007,
www.ilpungolo.com,
http://www.ilpungolo.com/leggi-tutto.asp?IDS=13&NWS=NWS5217
13) Stagnaro Sergio e Paolo Manzelli.
Semeiotica Biofisica Quantistica.
http://www.ilpungolo.com/leggi-tutto.asp?IDS=13&NWS=NWS5243
14) Stagnaro Sergio. Non Local
Realm. Response to Selection for Social Signalling
Drives the Evolution of Chameleon Colour Change. (01
February 2008).
www.plos.com,
http://biology.plosjournals.org/perlserv/?request=read-response&doi=10.1371/journal.pbio.0060025
I'dd appreciate your skilled answer.
Yours sincerely
By Sergio Stagnaro
>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>
To NCI Cancer gov.
Staff,
and to my friends of the list,
firstly many thanks for yur kind and frank answer, I
appreciate very much (See at bottom).
However, I underscore - once again - the present
urgent necessity to investigate both
ONCOLOGICAL Terrain (OT) and OT-dependent Inherited
Oncological Real Risk, recognized bedside rapidly
with a simple stethoscope, as do some Editors
in worthwhile manner.
In the exclusive interest of "mankind", we all must
state if these oncological concepts are FALSE
or TRUE, without further, distressing,
disheartening, disappointing DELAY (I can document
what I state, of course).
In a few words, ONCOLOGICAL Authorities, like NCI,
have to STATE now - and not tomorrow - if MY
OLD concepts as ONCOLOGICAL Terrain (OT) and
OT-dependent Inherited Oncological Real Risk have
been corroborated or falsify... In the later
case, obviously undermining HOW and Why, in my
PRESENCE, of course.
I state, once more, responsebly that without
ONCOLOGICAL Terrain (OT) and OT-dependent
Inherited Oncological Real Risk in UTERI CERVIX
cervical carcinoma is NOT possible, in spite
of type 16, and 18 HPV !
Before every study in this direction, we all,
INCLUDING peer-reviews "skilled" referees,
MUST know what "newborn-pathological, type I,
subtype a) ONCOLOGICAL, Endoarteriolar
Blocking devices really mean (For instance, read
Stagnaro Sergio. Newborn-pathological Endoarteriolar
Blocking Devices in Diabetic and Dislipidaemic
Constitution and Diabetes Primary Prevention. The
Lancet. March 06 2007.
http://www.thelancet.com/journals/lancet/article/PIIS0140673607603316/comments?totalcomments=1)
!
For precise information on this ovelooked, paramount
topic, See Bibliography,
Oncological Terrain,
a.s.o.
Let's all remember that, while we are writing KIND
and WISE mails, INNOCENT individuals,
apparently healthy... but positive for ONCOLOGICAL
Terrain (OT) and OT-dependent Inherited
Oncological Real Risk in some biological system,
are involved by CANCER, and .... sometimes... die,
too !
I am awaiting yours kind answer, and that of list
MEMBERS. Many Thanks to all of you.
Kind regards
By Sergio Stagnaro
>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>
----- Original Message -----
From: "NCI Cancer.gov Staff" <cancer.gov_staff@mail.nih.gov>
To: <dottsergio@semeioticabiofisica.it>
Sent: Tuesday, October 21, 2008 11:34 PM
Subject: RE: Cancer.gov Inquiry - HPV cause of
Cervical Cancer!
10/21/08_45321PIQ
Thank you for your e-mail to the National Cancer
Institute (NCI) regarding the human papillomavirus (HPV)
and cervical cancer. In your e-mail, you asked if
HPV can cause cervical cancer in women without
oncological terrain and inherited real risk. The
NCI, a component of the National Institutes of
Health, is the United States' principal agency for
cancer research. NCI e-mail staff are not familiar
with the term "oncological terrain." However, we
can offer information about HPV and cervical cancer
that you may find useful.
We encourage you to explore these resources:
--"PDQ(R) Treatment Summary for Health Professionals
on Cervical Cancer": This summary of information
from PDQ, the NCI's comprehensive cancer information
database, discusses treatment options for cervical
cancer and outlines treatment by stage. This
resource is available at
http://www.cancer.gov/cancertopics/pdq/treatment/cervical/healthprofessional/
on the Internet.
In particular, you may wish to refer to the
section on HPV infection and cervical cancer at
http://www.cancer.gov/cancertopics/pdq/treatment/cervical/HealthProfessional/page2
on the Internet.
--"Novel Genetic Test Predicts the Development of
Cervical Cancer":
This article on the NCI's Center for Cancer Research
Web site discusses a study that looked at the use of
a biomarker that would allow doctors to discern
lesions with low and high-risk for cervical cancer
disease progression. This article can be found at
http://ccr.cancer.gov/news/frontiers/january2007/pdfs/article_ried.pdf
on the Internet.
You will need Adobe Acrobat(r) Reader to view the
PDF (Portable Document Format) file mentioned above.
If you do not already have Adobe Acrobat Reader, you
can download the free software at
http://www.adobe.com/products/acrobat/readstep2.html
on the Internet.
You may also wish to perform a search of the medical
literature for published articles about HPV and
cervical cancer. The PubMed(R) database is a
service of the National Library of Medicine (NLM), a
component of the National Institutes of Health (NIH)PubMed
provides a free search tool to help people find
journal articles of interest in the health and
medical sciences. PubMed can be found at
http://www.pubmed.gov on the Internet.
We hope this information is helpful.
National Cancer Institute
NOTE: Should you get an error message when clicking
on the URL link(s) in this e-mail, you may have more
success opening the link if you copy and paste the
full URL into your browser window.
>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>
-----Original Message-----
From:
dottsergio@semeioticabiofisica.it
[mailto:dottsergio@semeioticabiofisica.it]
Sent: Monday, October 13, 2008 8:05 AM
To: NCI Cancer.gov Staff
Subject: Cancer.gov Inquiry - HPV cause of Cervical
Cancer !
Message: I ask if HPV can bring about cervical
Cancer also in women WITHOUT Oncological Terrain
"AND" Inherited Oncological Real Risk in Uterus
Cervix ?
By Sergio Stagnaro MD
P.S.
See and read:
Quantum Biophysical Semeiotics and Cancer Inherited
Real Risk in
www.nature.com:
http://blogs.nature.com/news/thegreatbeyond/2008/07/newsweak_darwin_vs_lincoln.html#comments
http://blogs.nature.com/nm/spoonful/2008/07/product_placement.html#comments
http://network.nature.com/forums/askthenatureeditor/1846?page=1#reply-5181
http://blogs.nature.com/nm/spoonful/2008/05/our_new_columns_narrowing_thehtml#comments
and specially
http://blogs.nature.com/nm/spoonful/2008/04/stress_as_a_therapy_1.html#comments
http://blogs.nature.com/news/thegreatbeyond/2008/07/new_drug_for_prostate_cancer_e.html#comments
http://blogs.nature.com/nature/journalclub/2008/02/nicholas_katsanis.html#comments
http://blogs.nature.com/nm/spoonful/2008/08/the_best_defense_against_biowa.html#comments
http://blogs.nature.com/news/thegreatbeyond/2008/08/bush_going_green_in_the_deep_b.html#comments
http://www.nature.com/news/2008/080828/full/news.2008.1068.html?q=2#last-comment
http://www.nature.com/news/2008/080902/full/news.2008.1076.html?q=2#last-comment
http://www.nature.com/news/2008/080917/full/news.2008.1116.html?q=2#last-comment
http://www.nature.com/news/2008/080923/full/news.2008.1124.html?q=2#last-comment
http://blogs.nature.com/news/thegreatbeyond/2008/10/election_watch.html#comments
IMPORTANTE:
Come Portale segnaliamo vari personaggi che hanno avuto
contrasti con le autorita' mediche, e per
essere precisi, affermiamo che
NON condividiamo in toto
le loro terapie (quelle
monoterapeutiche), in
quanto per noi, seguaci della
Medicina Naturale
la
malattia
(cancro compreso) e'
MULTIFATTORIALE, quindi
NESSUN prodotto puo', da solo, guarire dalla
malattia della quale si e' malati !
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